Conditions We Address

Erectile Dysfunction.

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What is it.

Erectile dysfunction (ED) is the persistent inability to achieve or maintain an erection sufficient for satisfactory sexual function. It affects approximately 30% of men over 40 and prevalence increases with age. Crucially, ED is an early marker of systemic vascular and metabolic disease.

Why does it happen.

Erection depends on nitric oxide (NO)-mediated vasodilation, the same endothelial mechanism that governs cardiovascular health. ED therefore reflects the same pathology: endothelial dysfunction, reduced blood flow, and impaired vascular responsiveness. Common drivers include: insulin resistance (damages endothelial function), hypertension (reduces vascular elasticity), low testosterone (reduces NO synthase activity), elevated cortisol (suppresses testosterone and vascular tone), elevated homocysteine (endothelial toxin), and smoking. ED precedes cardiovascular events by an average of 3–5 years — making it a critical preventive signal.

How AdeaHealth helps.

We run the full vascular, metabolic, and hormonal picture and address identified drivers. The cardiovascular and metabolic intervention that improves ED simultaneously reduces heart disease risk — a meaningful dual benefit.

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What we test and measure.

Total & free testosterone: NO synthase regulation and libido
ADMA & SDMA: endothelial dysfunction markers — inhibit nitric oxide production
Homocysteine: vascular endothelial damage
Fasting glucose, insulin, HbA1c: metabolic contribution to vascular health
ApoB, Lp(a), hs-CRP: cardiovascular risk

Prolactin: pituitary-driven testosterone suppression
Omega-3 Index: EPA/DHA support vascular function
Blood pressure & pulse: vascular function
PSA: baseline prostate health

What you can expect.

Improved erectile function through vascular, hormonal, and metabolic correction, often significant within 12–16 weeks. Parallel reduction in cardiovascular risk markers.